Protein C deficiency is genetic and can run in families. It is a protease that is part of the anticoagulant systems that are in place to keep coagulation (blood clot formation) in check. Excess clotting can occur when there is a mutation or change in one of the genes that code for the many factors that help blood to clot when necessary. This can lead to excess venous thrombosis (clotting).

Protein C deficiency is genetic and can run in families. It is a protease that is part of the anticoagulant systems that are in place to keep coagulation (blood clot formation) in check. Excess clotting can occur when there is a mutation or change in one of the genes that code for the many factors that help blood to clot when necessary. This can lead to excess venous thrombosis (clotting).

1)Thrombosis: Blood clot occurring in a blood vessel
2)Warfa rrin induced skin necrosis
3)Pur pura fulminans occurring especially in children, with hypotension, fever, and disseminated intravascular coagulation, usually following an infectious illness
4)Deep vein thrombosis: The formation of a thrombosis in the deep veins usually within the legs

Protein C deficiency is genetic and can run in families. It is a protease that is part of the anticoagulant systems that are in place to keep coagulation (blood clot formation) in check. Excess clotting can occur when there is a mutation or change in one of the genes that code for the many factors that help blood to clot when necessary

Protein C deficiency is genetic and can run in families. It is a protease that is part of the anticoagulant systems that are in place to keep coagulation (blood clot formation) in check. Excess clotting can occur when there is a mutation or change in one of the genes that code for the many factors that help blood to clot when necessary. This can lead to excess venous thrombosis (clotting).

Cirrhosis complete with coughing or vomiting of blood, jaundice, mental confusion and redness or inflamation along the effected segment of the viens